Journal of Neurology, Neurosurgery & Psychiatry
Fecha de publicación: 10 July 2020
DOI: http://dx.doi.org/10.1136/jnnp-2020-323226
Autores: Sergio Muñiz-Castrillo, Bastien Joubert, Mad-Hélénie Elsensohn, Anne-Laurie Pinto, Margaux Saint-Martin, Alberto Vogrig, Géraldine Picard, Véronique Rogemond, Valérie Dubois, Ryad Tamouza, Delphine Maucort-Boulch, Jérôme Honnorat
Background: Antibodies against contactin-associated protein-like 2 (CASPR2-Abs) have been described in acquired neuromyotonia, limbic encephalitis (LE) and Morvan syndrome (MoS). However, it is unknown whether these constitute one sole spectrum of diseases with the same immunopathogenesis or three distinct entities with different mechanisms.
